Congenital CMV Risk Stratifier
Timing · Long-term sequelae · Autism spectrum disorder · Fetal MRI interpretation
This tool stratifies the risk of long-term sequelae — including sensorineural hearing loss, neurodevelopmental impairment, and autism spectrum disorder (ASD) — based on the timing of primary maternal CMV infection and fetal MRI findings. It is built from the 2026 Leruez-Ville et al. prospective cohort study (Am J Obstet Gynecol, N=642, 23 years of follow-up, Necker-Enfants Malades Hospital, Paris).
Key findings this tool applies: all long-term CMV-related sequelae occurred exclusively after first-trimester maternal primary infection; ASD was diagnosed at a prevalence 4-fold higher than the general population (OR 4.25, 95% CI 1.63–21.33); and the absence of temporal lobe white matter abnormalities on fetal MRI carries a 100% negative predictive value for ASD. Every number displayed traces to a specific published source. View all references →
Key findings this tool applies: all long-term CMV-related sequelae occurred exclusively after first-trimester maternal primary infection; ASD was diagnosed at a prevalence 4-fold higher than the general population (OR 4.25, 95% CI 1.63–21.33); and the absence of temporal lobe white matter abnormalities on fetal MRI carries a 100% negative predictive value for ASD. Every number displayed traces to a specific published source. View all references →
Maternal Infection Profile
Classify and date the maternal CMV infection — timing is the single most important prognostic variable
Type of Maternal Infection
Neonatal Clinical Status
Symptomatic vs. asymptomatic status at birth is a key independent prognostic variable
Neonatal Presentation at Birth
All 11 children diagnosed with ASD in this cohort were symptomatic at birth. No ASD was diagnosed among asymptomatic neonates (p<0.001). ASD rate: 5.1% in symptomatic vs 0% in asymptomatic neonates.
Source: Leruez-Ville et al. AJOG 2026, Table 4 (N=642)
Source: Leruez-Ville et al. AJOG 2026, Table 4 (N=642)
Fetal / Postnatal MRI Findings
Temporal lobe white matter abnormalities are the neuroimaging signature linked to ASD risk
Three temporal lobe patterns to recognize (Figure 2, Leruez-Ville et al.): (1) T2 WM hyperintensity of temporal pole, (2) Temporal pole swelling/swollen appearance, (3) Temporal ventricular horn ectasia. Patterns 1 and 2 are associated with later ASD. Isolated ectasia was not associated with ASD in this cohort.
MRI Availability
Risk Profile & Clinical Guidance
Evidence-based assessment · Leruez-Ville et al. AJOG 2026 · N=642 · 23-year prospective cohort
Cohort Data Full Outcome Table by Infection Trimester
| Outcome | 1st Trimester N=288 | 2nd Trimester N=144 | 3rd Trimester N=72 | OR (1st vs 2nd+3rd) | p |
|---|---|---|---|---|---|
| Any Long-term CMV Sequelae | 76 (26.4%) | 0 | 0 | 155.88 (21.63–1123.45)* | <0.0001 |
| Sensorineural Hearing Loss | 46 (15.9%) | 0 | 0 | 83.04 (11.46–601.84)* | <0.0001 |
| Severe Imaging Findings | 16 (5.6%) | 0 | 0 | 26.20 (3.50–196.16)* | <0.0001 |
| Autism Spectrum Disorder | 5 (1.7%) | 0 | 0 | 8.39 (0.46–152.48)* | 0.07 |
| ADHD | 6 (2.1%) | 1 (0.7%) | 0 | 0.22 (0.05–1.86) | 0.25 |
| Language Impairment (no SNHL) | 2 (0.7%) | 1 (0.7%) | 0 | 0.67 (0.01–12.9) | 1.0 |
* Haldane-Anscombe correction. Source: Leruez-Ville M et al. AJOG 2026, Table 1.
Diagnostic Performance Temporal Lobe MRI → ASD Prediction
| Measure | Fetal MRI (N=204, ASD n=5) | Postnatal MRI (N=194, ASD n=11) |
|---|---|---|
| Sensitivity — WM Hyperintensity | 100% (95% CI 47.8–100) | 100% (95% CI 71.5–100) |
| Specificity — WM Hyperintensity | 91.9% (95% CI 87.3–95.3) | 77.6% (95% CI 70.8–83.4) |
| PPV — WM Hyperintensity | 23.8% (95% CI 16.3–33.3) | 21.1% (95% CI 17.0–26.0) |
| NPV — WM Hyperintensity | 100% (95% CI 98.0–100) | 100% (95% CI 97.4–100) |
| Sensitivity — WM + Swelling | 80.0% (95% CI 28.7–99.5) | 27.3% (95% CI 6.0–60.9) |
| Specificity — WM + Swelling | 97.5% (95% CI 94.2–99.2) | 97.8% (95% CI 94.5–99.4) |
| PPV — WM + Swelling | 44.4% (95% CI 23.6–67.9) | 42.8% (95% CI 16.0–74.6) |
Source: Leruez-Ville M et al. AJOG 2026, Table 3.